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Pompe disease (glycogenosis type II) is a rare lysosomal disorder caused by a mutational deficiency of acid α-glucosidase (GAA). This deficiency leads to glycogen accumulation in multiple tissues: heart, skeletal muscles, and the central nervous system. A knockout mouse model mimicking the human condition has been used for histological evaluation.



ebh 1e1sbe manual muscle

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Ebh 1e1sbe manual muscle
Article DOI https//doi.org/10.3201/eid2402.171074
ebh 1e1sbe manual muscle

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Annotation of ELM instances is based on manual annotation of the relevant literature, with a preference for instances whose functionality is thoroughly supported by multiple sources of complimentary experimental evidence.

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Pompe disease (glycogenosis type II) is a rare lysosomal disorder caused by a mutational deficiency of acid α-glucosidase (GAA). This deficiency leads to glycogen accumulation in multiple tissues: heart, skeletal muscles, and the central nervous system. A knockout mouse model mimicking the human condition has been used for histological evaluation.

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= blue highlighted text ends The second 60-day public review period will be held from November 1, 2018 through January 11, 2019. Public input can be submitted to the California Department of Education (CDE) via email or regular mail.

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AB, EBH and HK provided support in the design of the study. AGC and AB drafted the manuscript and all other authors contributed to the further writing of the manuscript and approved it. AGC and AB drafted the manuscript and all other authors contributed to the further writing of the manuscript and approved it.

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An isolated extracellular matrix-binding protein, designated as SdrE and its corresponding amino acid and nucleic acid sequences and motifs are described. The proteins, peptides, fragments thereof or antigenic portions thereof are useful for the prevention, inhibition, treatment and diagnosis of S. aureus infection and as scientific research tools.

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